If some patients may experience transient arrhythmia, the body will basically adjust itself and no treatment is needed.
On the other hand, electrical injuries may cause damage to the human heart in a more hidden state, making it difficult for clinical doctors to detect. In some cases of delayed death, the problem was not discovered until the autopsy was performed after the person died.
You think, if there is no abnormality in the electrocardiogram, the myocardial enzymes are not high, and the doctor cannot diagnose myocardial infarction, what treatment methods should the doctor use?
Please note that ST-segment elevation generally only indicates myocardial ischemia, not necessarily myocardial infarction.
Mr. Xie's argument not only directly points to the patient's myocardial infarction but also directly points to the very serious rupture of the "Ru" head muscle.
How can a practicing physician believe in a medical student's "fantasy"?
Luo Jingming was under the pressure of the other leader's gaze. Cao Zhao should know what he should say. He was from the National Association, and he knew his junior sister better than Cao Zhao and the others, and he believed in Xie more than Cao Zhao and the others.
Beep, beep.
The ECG fluctuations on the patient's ECG monitor showed that the patient's heart rhythm seemed to be heading towards arrhythmia. The ventricular rate reached more than 100 beats per minute. Children's heart rates can be relatively fast. However, this child is a big kid, and this heart rate is obviously abnormal. And there were premature beats in the ECG, which is definitely not a good sign.
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Cao Zhao stretched out his hand.
Pan, who was looked at by him, quickly handed over the electrocardiogram in his hand. No matter how much the man opposite him looked like the male protagonist of an idol drama, he was essentially a doctor like Pan, and he was a real technical master. He never wanted to be a movie star.
In fact, the ECG characteristics that Pan just reported are not wrong. The only problem is that children's conditions change quickly and heart disease patients themselves also change quickly.
Luo Jingming was so alert that he asked someone to push the defibrillator over and be ready to perform defibrillation and rescue at any time.
What exactly is wrong with the patient's heart rhythm? Is there something wrong with his skull muscles?
In the last surgery, I mentioned the Ru capitis, an internal structure of the heart. It is a myocardium connected to the tendons, and the end of the tendons is connected to the valve. It can be seen that the Ru capitis is connected to the valve through the tendons, which is like a mechanical conduction, providing a continuous biological power source for the opening and closing of the valve.
The left ventricular cervical muscle ultimately supports the most important valve of the heart, the mitral valve. Once it fails, it will inevitably affect the activity of the mitral valve, and the direct clinical symptoms will be acute pulmonary edema and heart failure caused by mitral regurgitation.
In fact, if the left ventricular Ru capitis muscle is completely ruptured, one-third of patients will die immediately, and more than half of them will not survive for more than 24 hours. If it is not corrected surgically, it can be said that death is inevitable.
"Are you talking about a partial fracture?" Cao Zhao asked the student for his opinion while scanning the electrocardiogram paper.
"Yes." Xie Wanying said, "Teacher, you know. The left ventricular craniocephalus muscle is divided into two groups. The anterior group is like a muscle integrated into one piece in anatomy. Once it breaks, it will be a complete break, and the patient is prone to sudden death. The posterior group is not like that. It is a combination. When it breaks, it is difficult to break completely and usually manifests as a partial break. The electrocardiogram corresponding to the anterior group of craniocephalus muscles appears on the anterior wall, and the electrocardiogram of the posterior group shows the inferior wall."
This corresponds exactly to the electrocardiogram characteristics of inferior wall myocardial ischemia mentioned by classmate Pan.
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